In contrast XX gonadal dysgenesis has a normal female chromosome situation.
2.
Women with 46 XX gonadal dysgenesis experience primary amenorrhea with hypergonadotropic hypogonadism.
3.
Although similar in some ways to mixed gonadal dysgenesis, the conditions can be distinguished histologically.
4.
At this point it is usually possible for a physician to make a diagnosis of XX gonadal dysgenesis.
5.
Abnormal gonads ( due to gonadal dysgenesis and androgen insensitivity syndrome ) have a high risk of developing a dysgerminoma.
6.
When the Y fragment is minimal and nonfunctional, the child is usually a girl with the features of Turner syndrome or mixed gonadal dysgenesis.
7.
The magnitude of the testosterone increase can help differentiate between androgen resistance and gonadal dysgenesis, as does evidence of a uterus on ultrasound examination.
8.
In Turner syndrome there is a demonstrable abnormality in or absence of one of the sex chromosomes that is the cause of the development of gonadal dysgenesis.
9.
There are forms of 46 xx gonadal dysgenesis wherein abnormalities in the FSH-receptor have been reported and are thought to be the cause of the hypogonadism.
10.
Testicular function can also be assessed by measuring serum anti-M�llerian hormone levels, which in turn can further differentiate PAIS from gonadal dysgenesis and bilateral anorchia.
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